Most infectious diseases have a sexually dimorphic outcome. We investigate the reasons for this not only with a host-centered view but also by considering that pathogens adapt to the most commonly encountered sex.
One of the parasite strategy to increase its transmission is to manipulates its host. I started science by studying such strategy.
Infection outcome depends on the success of the parasite within its host. With theoretical and empirical approaches, we study what influences pathogen dynamics and its implication on symptoms.
Parthenogenetic species produce mainly daughter without males. We study the selection during the rare event of sexual reproduction.
Parasites need to go through several infection steps to be successfull. We study how the evolution at each step shapes host-parasite coevolution.
Genetic variation is the raw material for evolution. We pertain to identify, using GWAS and the Drosophila Reference Genetic Panel (DGRP), the genetic basis of various quantitative traits (e.g. phenotypic plasticity, insecticide resistance).
Lyme disease is a major threat in many countries. The bacteria responsible for it is mainly transmitted by ticks feeding on mammals. We studied the role of seabirds as reservoir.