Most infectious diseases have a sexually dimorphic outcome. We investigate the reasons for this not only with a host-centered view but also by considering that pathogens adapt to the most commonly encountered sex.
Infection outcome depends on the success of the parasite within its host. With theoretical and empirical approaches, we study what influences pathogen dynamics and its implication on symptoms.
Parthenogenetic species produce mainly daughter without males. We study the selection during the rare event of sexual reproduction.
Genetic variation is the raw material for evolution. We pertain to identify, using GWAS and the Drosophila Reference Genetic Panel (DGRP), the genetic basis of various quantitative traits (e.g. phenotypic plasticity, insecticide resistance).